Frequently, after exposure to Borrelia burgdorferi, the primary pathogen that causes Lyme and after the antibiotic therapy regime, we see an alteration in the person's innate immune response. This is something that can cause what is referred to as, "Lyme Syndrome." In this scenario, one or more aspects of the immune defense and repair system are impaired.
The result of this impairment is that the body is unable to destroy the pathogens that are infecting it (Ehrlichia, Babesia and Richettsia have been found in conjunction with B. burgdorferi in LD). These pathogens travel freely in the bloodstream, establish themselves in hospitable tissues and trigger a number of chronic, sometimes severe symptoms: joint pain, central nervous system symptoms, digestive tract symptoms and even cardiovascular symptoms.
Many clinicians are making a distinction between LD and LS, the latter being a less-precise and far-reaching diagnosis. We see with LS a multi-system, inflammatory, impaired-repair syndrome. As a result, LS frequently becomes an auto-immune syndrome. It often is clinically overlapped by other immune impaired-repair syndromes such as SLE, Thyroiditis, MS, RA, Diabetes and Sjogren's.
The immune memory cells (B and T cell lymphocytes) then often become reactive not only against the original pathogens (antigens) but against the host antigens (self). This then results in the LD (an infectious disease) transitioning into LS (an autoimmune disease) mostly because of the phenomenon of the cross-reactive immune response, something I talked about in an earlier blog.
Once the body shows signs of LS, it is vital that it be correctly nutritionally suppported, including supporting both parts of the immune system: the gut and the adrenals/spleen/liver complex, as well as all the other affected areas.
This information is gleaned from: The Journal of Neuroimmunology and Nature Medicine.
Dr. Esther
drkollars@gmail.com
fixdhealthcare.com
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